Mainly, urea and uric acid producing even more ammonia.

同时尿素和尿酸会产生更多的氨。

Uric acid has limited solubility in body fluids, though.

然而，尿酸很难溶于体液中。

The fatty acids, urea, uric acid and lactate in the sebaceous film protect and hydrate the skin.

皮脂膜中的脂肪酸、尿素、尿酸和乳酸对皮肤有着保护和滋润作用。

These stones, also like uric acid stones, are red-brown in color and radiolucent under an X-ray.

这类结石也像尿酸结石一样，呈红棕色，X射线可透过。

Since uric acid is a breakdown product of purines, a very common reason for high levels of uric acid is consuming lots of purines.

于尿酸是呤的分解产物，故高尿酸是大呤的结果。

What that stuff is is uric acid.

AVIR：那西就是尿酸。

Finally, there are some rare stones made of xanthine, which, just like uric acid, is a byproduct of purine breakdown.

最后，还有呤形成的罕见结石，同尿酸一样，是呤分解的副产物。

Uricosuric medications, like probenecid, increase excretion of uric acid by the kidneys.

促尿酸排泄药物，如丙磺舒，会提高尿酸的肾脏排泄。

There are also medications that help to decrease uric acid levels, which include xanthine oxidase inhibitors like allopurinol.

也有一些药物可以帮助降低尿酸含，包括呤氧化酶抑制剂，如别醇。

But look at my uric acid level.

但是看看我的尿酸水平。

My uric acid has now gone back to normal.

我的尿酸现在已经恢复正常了。

Less common kidney stones are made of calcium phosphate, or uric acid.

不太常见的肾结石是磷酸钙或尿酸制成的。

Other nitrogenous wastes include uric acid and creatinine.

其他含氮废物包括尿酸和肌酐。

When the birds feed on tainted carcasses, uric acid crystallizes in their kidneys.

当鸟类以受污染的尸体为食时，尿酸会在它们的肾脏中结晶。

Urine contains compounds that consist of calcium, sodium, potassium, oxalate, uric acid, and phosphate.

尿液中含有钙、钠、钾、草酸盐、尿酸和磷酸盐组成的化合物。

It happens when uric acid crystals form in the joints, causing them to swell.

当关节中形成尿酸结晶并导致关节肿胀时， 就会发生这种情况。

The most common type of stones are calcium oxalate stones, though other types include uric acid stones and struvite stones.

草酸钙结石是最常见的类型，其他类型的结石有尿酸结石和感染性结石。

Xanthine oxidase is an enzyme involved in the breakdown of purines to uric acid, so inhibiting this enzyme results in less uric acid production.

呤氧化酶负责分解呤为尿酸，所以抑制这个酶，就可以减少尿酸的生成。

Also, some individuals have a genetic predisposition to overproduction of uric acid while others with chronic kidney disease may be unable to excrete the uric acid.

同时，某些个体遗传易感，倾向于过生成尿酸；而慢性肾病病人因其无法正常排泄尿酸，也可能诱发痛风。

At a physiologic pH, uric acid loses a proton and becomes a urate ion, which then binds sodium, forming monosodium urate which crystallizes and ultimately forms uric acid stones.

生理PH值下，尿酸失去一个质子变成尿酸盐离子，随之与钠结合，形成单钠尿酸盐，结晶后最终形成尿酸结石。